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doi: 10.1038/ni1460 173

In spite of the identification of these mitochondrial protein targets, a direct effect of their modification on compromising mitochondrial function is not evident, and some of these changes could be consequences rather than causes of mitochondrial dysfunction

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The review situates Dihexa within the renin-angiotensin systems central nervous system arm, summarizes the HGF/c-Met activation hypothesis, and surveys behavioral pharmacology in rodent cognitive models